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specific elisa kits for mouse tnf-α, il-1β, il-6, ccl-2 and il-10  (Thermo Fisher)


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    Thermo Fisher specific elisa kits for mouse tnf-α, il-1β, il-6, ccl-2 and il-10
    Specific Elisa Kits For Mouse Tnf α, Il 1β, Il 6, Ccl 2 And Il 10, supplied by Thermo Fisher, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/mouse-specific+il-6+elisa/pmc11579449-90-10-19
    Average 90 stars, based on 1 article reviews
    specific elisa kits for mouse tnf-α, il-1β, il-6, ccl-2 and il-10 - by Bioz Stars, 2026-09
    90/100 stars

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    Related Articles

    other:

    Article Title: Rbm10 regulates inflammation development via alternative splicing of Dnmt3b.
    Article Snippet: RNA-binding motif 10 (Rbm10) is an RNA-binding protein that regulates alternative splicing, but its role in inflammation is not well defined.. Here, we show that Rbm10 controls appropriate splicing of DNA (cytosine-5)-methyltransferase 3b (Dnmt3b), a DNA methyltransferase, to regulate the activity of NF-κB-responsive promoters and consequently inflammation development.. Rbm10 deficiency suppressed NF-κB-mediated responses in vivo and in vitro.

    Article Title: Computer model of IL-6-dependent rheumatoid arthritis in F759 mice.
    Article Snippet: The interleukin-6 (IL-6) amplifier, which describes the simultaneous activation of signal transducer and activator of transcription 3 (STAT3) and NF-κb nuclear factor kappa B (NF-κB), in synovial fibroblasts causes the infiltration of immune cells into the joints of F759 mice.. The result is a disease that resembles human rheumatoid arthritis.. However, the kinetics and regulatory mechanisms of how augmented transcriptional activation by STAT3 and NF-κB leads to F759 arthritis is unknown.

    Article Title: Role of Chondrocytes in the Development of Rheumatoid Arthritis Via Transmembrane Protein 147-Mediated NF-κB Activation.
    Article Snippet: Mouse cells are responsive to human IL-6 which allowed us to measure mouse IL-6 in the culture supernatant after stimulation with human IL-6 and mouse IL-17A using mouse-specific IL-6 ELISA (eBioscience and BD Biosciences, Tokyo, Japan).



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